Ukuhogela ubuthi

Uhlobo oluvame kakhulu lokudakwa ngokweqile ekwenzeni ukwelashwa luholela ekufakeni ubuthi. Ngisho nabantu abangahlotshaniswa nomsebenzi wokukhiqiza noma ukusetshenziswa kwalesi sidakamizwa bavame ukuvezwa kule ngozi ngokufaka uthuli uthuli noma imfucuza.

Izimpawu nezibonakaliso zokuhogela ubuthi

Ukubonakaliswa kwemitholampilo yenkinga ehlongozwayo kuhlotshaniswa nokwanda kwensimbi encane egazini (okungaphezu kuka-800-100 μg / L we-fluid biological). Uketshezi olunamandla ngamapuphu okuhola noma uthuli oluqukethe lesi sakhi samakhemikhali luhambisana nalezi zimpawu:

Kubuye kune-poclinical chronic lead lead, lapho ukugxila kwensimbi egazini kukhula kancane kancane ngezikhathi zokunciphisa okungakahleleki. Njengomthetho, leli fomu lesi sifo lihlukile kubantu abasebenza ezimbonini zamakhemikhali besebenzisa ama-lead compounds. Unayo izimpawu ezilandelayo:

Ukwelashwa kwe-poisoning eholayo

Okokuqala, susa umthombo wokuxhumana ngamaphunga nothuli olune-lead. Ukwelashwa okuqhubekayo kusekelwe ekusungulweni komzimba wezidakamizwa ezakha ama-chemical complexes ngensimbi - izimboni ezivimbela umphumela omubi wezinto futhi zifaka isandla ekuqothulweni kwayo. Ukuze wenze lokhu, i-calcium usawoti dimercaprol, i-EDTA ne-cuprin (D-penicillamine) isetshenziswa, futhi i-agent entsha, i-dimercaptosuccinic acid, ibuye ihlolwe.

Ukwenza ngcono ukwelashwa kunqunywe ngemuva kokuqhathanisa inani lemithi ejojowe kanye noholayo, okuxhunyiwe emcinini. Ukwelapha kubhekwa njengempumelelo uma i-1 mg ye-akhawunti yezidakamizwa eziyinkimbinkimbi ingaphezu kuka-1 μg wokuhola emcibini okhishwe emini.

Nge-poisoning eqinile nokuthuthukiswa kwe-encephalopathy enzima, kunconywa ukuphatha izidakamizwa ezimbili ngesikhathi esisodwa - usawoti we-calcium we-EDTA ne-Dimercaprol. Ukuba khona kwe -edema ye- cerebral kuhlanganisa nokusetshenziswa kweDexamethasone ne-Mannitol.

Ukwelashwa okugcwele izinsuku ezingu-5, kodwa uma kunesidingo, kungabuyekezwa emva kwekhefu elifushane.